Healthy cell respiration in the mitochondriais the basis of health

The first disease due to mitochondrial dysfunction wasdescribed in 1962 by Luft and coworkers in the Journalof Clinical Investigation. Since then, there has been adramatic increase in interest and research into the struc-ture and function of the mitochondria and in knowledge of their metabolic significance and causal relationships with numerous health impairments. Depending on their energy demand, organs are affected to differing degrees by an impairment of mitochondrial function. The heart  and skeletal muscle cells, brain and retina therefore show a much more pronounced response to mitochondrial insufficiency.


Mitochondrial dysfunction is characterised by reduced  ATP synthetic performance and an associated energy deficit on the one hand, and by increased oxidative stress and greater mutation of mitochondrial DNA (mtDNA) on the other. This leads to functional insufficiency of the affected cells and tissues and to their premature death and mutagenic change. Muscle weakness, rapid muscle fatigability, poor training capacity or muscle pain on exercise and even at rest can be initial signs of mitochondrial dysfunc-tion in young patients.

Mitochondrial diseases arising from mtDNA mutationshave a prevalence of approximately 10 - 20 per 100,000. Today, more than 200 mutations of mtDNAare known. Besides the diseases caused by a hereditarygenetic defect in the mtDNA, however, an increasing  number of lifetime acquired diseases are now being seen as causally associated with mitochondrial dysfunc-tion. These include neurodegenerative diseases such as Alzheimer’s or Parkinson’s disease, cardiovascular  diseases, diabetes mellitus or malignant conditions.

To function properly, these antioxidant systems need thebioactive substances of the kind contained in EnzymeYeast Cells Dr. Wolz®. Human genetic material is 70 %identical to the genetic material of the enzyme yeast cells.All the ingredients of Enzyme Yeast Cells Dr. Wolz® therefore show good bioavailability.


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